This is only the first two pages of the actual issue

The Coming Food Shortage! Part 2

We need to report a few more pieces of information concerning the coming food shortage around the world. The Nutrition Action Healthletter, June 2001, had a cover story concerning Mad Cow Disease. 90% of the newsletters information was erroneous. For instance, one statement said: "Mad Cow Disease has NEVER been found in the U.S."

That statement was simply NOT true. My father-in-law worked at a meat packing company in Springfield, Missouri, and personally saw cows with Downer Cow Syndrome - which later was identified as BSE - bought, cut up, and shipped to local restaurants. However, one section of this report may have something of value. It was entitled:

A METHIONINE TO THEIR MADNESS

"Why do some people who eat BSE-infected meat develop variant Creutzfeldt-Jakob Disease while others don't seem to? To find out, scientists have tested tissue samples from 76 of the 101 people who have died from vCJD. Remarkably, all 76 `were homozygous for methionine at codon 129 on the prion protein gene,' as the National Institutes of Health's Paul Brown puts it.

"Translation: they inherited from each of their parents a gene that substitutes one amino acid (methionine) for another (valine) in one portion of the prion protein that the gene tells the body to make. For some reason, `the methionine form of the protein seems to make people more susceptible to vCJD,' says Brown.

"Four out of every ten people inherit a copy of the gene from each of their parents. It's always possible that people who don't have two copies of the gene also develop vCJD, but that `it just takes them longer to become ill.' cautions Brown."

[Editor's note: This is the same National Institutes of Health that sold scrapie sheep to sheep farms across America in the 1970s.] There certainly seems that more causes could be involved with the Mad Cow Disease [BSE] and its human form [CJD] than meets the eye. Let's invest-igate a little more!

ICI's ex-chemical weapon insecticide causes BSE & CJD

Cover-up Insecticide causes Mad Cow disease

by Fintan Dunne

Research Kathy McMahon (13th December 2000)

Pharmaceutical interests in the UK [Britain] are ignoring new scientific research that shows the insecticide used in the UK government's own warble-fly campaigns triggered the UK surge of `Mad Cow' disease.

The new work shows that prions can BOND with manganese in animal feeds OR mineral licks. These manganese prions can cause the neurological degeneration seen in BSE. And other researchers unpublished work shows that prions in the bovine [cow] spine - along which insecticides are applied - can be damaged by ICI's Phosmet organophosphate (OP) insecticide - CAUSING the disease.

By a similar process, prions in human brains can be damaged by lice lotions containing organophosphate. Later in life, this can result in neurological diseases like CJD and Alzheimer's.

British scientists have led the current theory that an infectious prion in bonemeal fed to cattle causes bovine spongiform disease (BSE). Infectious prions are also claimed to cause new variant Creutzfeld-Jacob Disease (CJD) in humans - from ingesting beef. But the infectious prion theory serves to [help] obscure a tragic chemical poisoning scandal behind the majority of BSE cases.

Latest experiments by Cambridge University prion specialist, David R. Brown, have shown that manganese bonds DESTRUCTIVELY with prions. Many might be sur-prised to hear that organophosphates were developed by NAZI CHEMISTS during the course of World War Two, as a CHEMICAL WEAPON NERVE AGENT.

The marginalised research has devastating financial implications for ICI. It would provide a firm basis for litigants [filing of law suits] - who could include CJD sufferers, farmers across the world and families of the many British farmers who committed suicide during this BSE debacle.

Phosmet organophosphate has been used at high doses in British warble fly campaigns. In 1996, former ICI subsidiary Zeneca sold the phosmet patent to a PO Box company in Arizona called Gowan - just one week BEFORE the UK government admitted to a link between BSE and nvCJD.

The politically well-connected British pharmaceut-icals group, ICI has the clout to influence research into any cause OTHER than the infective model. Indeed no substantive alternative research has been done. British BSE disease management and research bodies have taken decisions that do not seem guided by spirited scientific enquiry. Mysterious prions that jump species is the preferred research arena.

Scientist and organic farmer, Mark Purdey gave evidence to the UK BSE inquiry, that warble fly insecticide was the [or one of] the cause[s] of the disease. The scientist wheeled out to rubbish Purdy's evidence. Dr. David Ray, later turned out to have been receiving funding from the insecticide manufacturer ICI.

A lobby group that included Bayer, Monsanto, Novartis, Pfizer, Roche and Schering-Plough was behind the effort to discredit Purdey. In December 1999, the same David Ray was appointed to the UK Veterinary Products Committee (VPC) - a government body that licenses animal medicines.

Purdey has been consistently denied even exploratory funding to extend his privately supported research. Yet the Purdey/Brown chemical poisoning model matches with the epidermiological spread of CJD clusters in humans. It also predicts the incidence of BSE-type diseases in animals. The accepted infectious model fits neither.

The pharmaceutical industry is all the more deter-mined to hide the chemical source of BSE and CJD, be-cause a spotlight on chemicals would expose the role the insecticides have in Alzheimer's - another neuro-degenerative disease - that might lead to [legal] claims which would dwarf those from BSE and CJD litigants [lawsuits]. In fact, two leading brain researchers into CJD and Alzheimers have died in suspicious circumstances in recent years.

In the United States, the Environmental Protection Agency is already reviewing Phosmet's safety. The Centers for Disease Control in the U.S. has recently conducted experiments on mice that confirm the organophosphate risk.

This Article's Author and Researcher's Thoughts on European Community Cattle Slaughter to End BSE

Not only is the European Community beef slaughter campaign futile - because BSE disease is mostly non-infectious, but unless the underlying CHEMICAL cause is addressed, BSE will simply reappear from chemical causes. A new warble fly campaign is already underway in France using the organophosphate insecticide.

Of greater concern is that some lotions for
scabies and head lice are now priming children and adults for CJD and Alzheimer's in later life.

BONDING THE PRION

Cambridge University prion biochemist, David R. Brown, is dismissive of the science behind the infectious model of BSE. He terms it "a very limited amount of science by a few assumed-reputable scientists." He insists there is "no evidence an infectious agent is present in either meat or milk." [Editor's note: I feel obligated to give an alternative viewpoint not being a scientist. It seems scientists cannot agree among themselves WHY BSE and CJD are occurring.]

"Simple tests on udder walls of cows - which could easily detect an infectious prion - have not been done, why I don't understand." [Editor's note: If prion proteins do not produce antibodies, how could the infectious prion be so easily detected?]

A number of researchers have found that organophosphate (OP) in systemic warble fly insecticide CAN DEFORM THE PRION MOLECULE, rendering it ineffective at buffering free radical effects in the body. Worse still, the prion is then partial to BOND WITH MANGANESE and become a "rogue" prion. A chain reaction whereby rogue prions turn others to rogues also, can explain the bovine spongiform disease mechanism.

Brown showed how prion protein bonds benignly with copper, but lethally with manganese. Even natural variations in relative environmental availability of manganese versus copper can trigger prion degradation.

The CJD and BSE symptoms mirror manganese madness, an irreversible fatal neuro-psychiatric degenerative syndrome that plagued manganese miners in the first half of the last century.

SHINING A LIGHT ON SPONGIFORM

Organic dairy farmer and ...independent scientist, Mark Purdey, says the accepted theory of transmission from BSE-infected cattle to human CJD by bonemeal or meat is dependent on a mutant prion that has never been isolated under the scientific protocol called Koch's postulates.

Purdey's insistence on sticking to the letter of this scientific law earned him the condemnation of UK [British] officialdom when he first mooted [presented] his theory. But Purdey pointed to CJD clusters downwind of a British Phosmet production plant to back his case.

He gave evidence to the UK Government BSE inquiry and was supported by Conservative MP, Thessa Gorman. His views were discounted, but his subsequent re-search and the new Cambridge prion work have confirmed (continued...)